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Cattle Boards
NCBA, R-CALF, COOL, USDA (No Politics!)
Cattle EK211 PRNP polymorphism susceptible to H-type BSE agent from either E211K or wild type donors after oronasal inoculation
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<blockquote data-quote="TexasJerseyMilker" data-source="post: 1835543" data-attributes="member: 42782"><p>. . . . Respectfully, Nobody cares.</p><p></p><p>There is classical mad cow disease and atypical mad cow disease. Both strains of atypical L-BSE and</p><p>H-BSE rarely show up in old cutter and canner cows at slaughter, 6 times out of millions of cattle since 2003. Atypical mad cow is so uncontagious researchers have to inject it directly into the brains of experimental animals.</p><p></p><p><a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6989211/" target="_blank">https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6989211/</a> </p><p>Conclusion</p><p>"Transmission experiments and subpassages of both atypical BSE forms into laboratory rodents revealed that the phenotypes of both atypical BSE strains are not fully stable. This has been interpreted as a possible explanation for the origin of C-BSE which might have emerged after passages of one of the atypical BSE strains. However, a transmission from L-BSE or H-BSE to C-BSE has never been observed after transmission to cattle or sheep, rendering this assumption rather unlikely."</p></blockquote><p></p>
[QUOTE="TexasJerseyMilker, post: 1835543, member: 42782"] . . . . Respectfully, Nobody cares. There is classical mad cow disease and atypical mad cow disease. Both strains of atypical L-BSE and H-BSE rarely show up in old cutter and canner cows at slaughter, 6 times out of millions of cattle since 2003. Atypical mad cow is so uncontagious researchers have to inject it directly into the brains of experimental animals. [URL]https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6989211/[/URL] Conclusion "Transmission experiments and subpassages of both atypical BSE forms into laboratory rodents revealed that the phenotypes of both atypical BSE strains are not fully stable. This has been interpreted as a possible explanation for the origin of C-BSE which might have emerged after passages of one of the atypical BSE strains. However, a transmission from L-BSE or H-BSE to C-BSE has never been observed after transmission to cattle or sheep, rendering this assumption rather unlikely." [/QUOTE]
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Cattle Boards
NCBA, R-CALF, COOL, USDA (No Politics!)
Cattle EK211 PRNP polymorphism susceptible to H-type BSE agent from either E211K or wild type donors after oronasal inoculation
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